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Lipopolysaccharide exposure during late embryogenesis triggers and drives Alzheimer‐like behavioral and neuropathological changes in CD‐1 mice

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Infections could contribute to Alzheimer's disease (AD) neuropathology in human. However, experimental evidence for a causal relationship between infections during the prenatal phase and the onset of AD is lacking. Click to show full abstract

Infections could contribute to Alzheimer's disease (AD) neuropathology in human. However, experimental evidence for a causal relationship between infections during the prenatal phase and the onset of AD is lacking.

Keywords: embryogenesis triggers; drives alzheimer; late embryogenesis; triggers drives; exposure late; lipopolysaccharide exposure

Journal Title: Brain and Behavior
Year Published: 2020

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