LAUSR

Epicardial adipose tissue (EAT) plays important physiological and pathological roles in the regulation of myocardial function. By sharing the same microcirculation as the myocardium, EAT may maintain cardiac health via mechanical, metabolic, thermogenic and paracrine functions1; on the other hand, EAT inflammation in obesity may mediate myocardial dysfunction and heart failure (HF).1 Prior studies reported conflicting associations between EAT and HF (Table 1).2–6 Importantly, the method of measuring EAT may impact results. Magnetic resonance imaging (MRI) allows measurement of whole heart EAT volume or mass, whereas