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Interaction of receptor for advanced glycation end products (RAGE) with amyloid‐β increases amplification of oxidative stress and plays pathological roles in Alzheimer's disease (AD). Oxidative stress leads to α‐synuclein aggregation… Click to show full abstract
Interaction of receptor for advanced glycation end products (RAGE) with amyloid‐β increases amplification of oxidative stress and plays pathological roles in Alzheimer's disease (AD). Oxidative stress leads to α‐synuclein aggregation and is also a major contributing factor in the pathogenesis of Lewy body dementias (LBDs). Therefore, we aimed to investigate whether RAGE gene polymorphisms were associated with AD and LBDs.
Keywords:
alzheimer disease;
lewy body;
body dementias
Journal Title: International Journal of Geriatric Psychiatry
Year Published: 2017
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Journal Title: International Journal of Geriatric Psychiatry
Year Published: 2017
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!