Defects in the α‐synuclein, leucine‐rich repeat kinase 2, or glucocerebrosidase genes have been regarded as essential contributors to PD. However, genetic variability of these genes with respect to early‐onset PD… Click to show full abstract
Defects in the α‐synuclein, leucine‐rich repeat kinase 2, or glucocerebrosidase genes have been regarded as essential contributors to PD. However, genetic variability of these genes with respect to early‐onset PD remains poorly defined for the Chinese demographic.
               
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