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Prenatal bisphenol a exposure leads to reproductive hazards on male offspring via the Akt/mTOR and mitochondrial apoptosis pathways

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Bisphenol A (BPA) exposure is ubiquitous, and in laboratory animals and humans, exposure has been associated with male spermatogenesis dysfunction. However, it is largely unknown if this association has a… Click to show full abstract

Bisphenol A (BPA) exposure is ubiquitous, and in laboratory animals and humans, exposure has been associated with male spermatogenesis dysfunction. However, it is largely unknown if this association has a fetal origin.

Keywords: bisphenol; bisphenol exposure; exposure; leads reproductive; prenatal bisphenol; exposure leads

Journal Title: Environmental Toxicology
Year Published: 2017

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