LAUSR

The motor symptoms of Parkinson’s disease (PD) are predominantly due to progressive degeneration of nigral dopaminergic neurons. In most cases, there is a substantial asymmetry of clinical symptoms from disease onset [1]. Such lack of symmetry in the appearance of symptoms is so conspicuous that it serves as a clinical parameter to differentiate the disease from other neurodegenerative parkinsonian syndromes. Hemichorea is a hyperkinetic movement disorder that is provoked by unilateral basal ganglia dysfunction, usually manifested by structural lesions or metabolic derangements [2, 3]. Most putatively related anatomical structures are lentiform nucleus, thalamus, and subthalamic nucleus [2]. PD and hemichorea are similar in terms of asymmetrically malfunctioning brain structures contralateral to clinical manifestations. We observed a PD patient with rare phenomenon of hemichorea.