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Sickness in mammals can lead to cognition deficits, although the underlying mechanisms remain elusive. In a recent Nature Medicine article, Garré et al. (2017) report that sickness-induced cortical dendritic spine loss… Click to show full abstract
Sickness in mammals can lead to cognition deficits, although the underlying mechanisms remain elusive. In a recent Nature Medicine article, Garré et al. (2017) report that sickness-induced cortical dendritic spine loss and impaired memory formation is mediated by CX3CR1+ monocyte-derived TNF-α.
Keywords:
medicine;
cx3cr1 monocytes;
response infection;
behavior cx3cr1;
spineless behavior;
monocytes response
Journal Title: Immunity
Year Published: 2017
Link to full text (if available)
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Journal Title: Immunity
Year Published: 2017
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!