As reported by Smyrnias et al. [(1)][1], pre-induction of mitochondrial unfolded protein response (UPRmt) using nicotinamide riboside is sufficient to prevent cardiac dysfunction induced by chronic hemodynamic overload in rodents. These… Click to show full abstract
As reported by Smyrnias et al. [(1)][1], pre-induction of mitochondrial unfolded protein response (UPRmt) using nicotinamide riboside is sufficient to prevent cardiac dysfunction induced by chronic hemodynamic overload in rodents. These findings open up a new avenue for strategies targeting
               
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