Anti-N-methyl-d-aspartate (NMDA) receptor encephalitis was first described in 2007. The most frequent forms result from extracellular antibody-mediated autoimmunity. Prognosis improves with early, aggressive immunotherapy; aetiological diagnosis is therefore essential. Diagnosis… Click to show full abstract
Anti-N-methyl-d-aspartate (NMDA) receptor encephalitis was first described in 2007. The most frequent forms result from extracellular antibody-mediated autoimmunity. Prognosis improves with early, aggressive immunotherapy; aetiological diagnosis is therefore essential. Diagnosis is based on the detection of anti-NMDA receptor antibodies in the serum and cerebrospinal fluid (CSF). MRI usually shows normal findings or mild alterations. Given the lack of specificity and the frequently subacute onset of symptoms, brain
               
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