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Effector T-cell contraction is one of the primary means by which immune homeostasis in the peripheral tissues is reestablished following an inflammatory response. In this issue of Mucosal Immunology, Hashimoto-Hill… Click to show full abstract
Effector T-cell contraction is one of the primary means by which immune homeostasis in the peripheral tissues is reestablished following an inflammatory response. In this issue of Mucosal Immunology, Hashimoto-Hill et al.1 show that retinoic acid (RA) transcriptionally upregulates P2X7 primarily in effector T cells of the intestine sensitizing them to NAD-induced cell death (NICD); thus, demonstrating a previously unrecognized role of RA in effector T-cell contraction.
Keywords:
acid takes;
effector cells;
retinoic acid;
effector;
immunology
Journal Title: Mucosal Immunology
Year Published: 2017
Link to full text (if available)
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Journal Title: Mucosal Immunology
Year Published: 2017
Link to full text (if available)
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recommendations!