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A computationally designed small molecule inhibits the replication of prion-protein aggregates and ameliorates neurological symptoms in mouse and non-human-primate models of transmissible spongiform encephalopathy. Click to show full abstract
A computationally designed small molecule inhibits the replication of prion-protein aggregates and ameliorates neurological symptoms in mouse and non-human-primate models of transmissible spongiform encephalopathy.
Keywords:
prion diseases;
designer chaperone;
prion;
chaperone prion
Journal Title: Nature Biomedical Engineering
Year Published: 2019
Link to full text (if available)
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Journal Title: Nature Biomedical Engineering
Year Published: 2019
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!