Although cognitive dysfunction persists through affective and euthymic states in bipolar disorder (BD), its neurobiological correlates remain undetermined. We explore whole‐cortex intracortical myelin (ICM) and cognition in BD‐I and controls. Click to show full abstract
Although cognitive dysfunction persists through affective and euthymic states in bipolar disorder (BD), its neurobiological correlates remain undetermined. We explore whole‐cortex intracortical myelin (ICM) and cognition in BD‐I and controls.
               
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