Premature birth is a common obstetric complication but its pathogenesis is unclear. Inflammation at the maternal‐fetal interface in preterm labor leads to the infiltration of neutrophils, which promotes inflammatory responses… Click to show full abstract
Premature birth is a common obstetric complication but its pathogenesis is unclear. Inflammation at the maternal‐fetal interface in preterm labor leads to the infiltration of neutrophils, which promotes inflammatory responses and induces the degradation of extracellular matrix and cell apoptosis, thus contributing to preterm labor. It is unclear whether neutrophil extracellular traps (NETs), a functional form of neutrophils, are involved in preterm labor.
               
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