LAUSR.org creates dashboard-style pages of related content for over 1.5 million academic articles. Sign Up to like articles & get recommendations!

NETs promote ROS production to induce human amniotic epithelial cell apoptosis via ERK1/2 signaling in spontaneous preterm birth

Photo from wikipedia

Premature birth is a common obstetric complication but its pathogenesis is unclear. Inflammation at the maternal‐fetal interface in preterm labor leads to the infiltration of neutrophils, which promotes inflammatory responses… Click to show full abstract

Premature birth is a common obstetric complication but its pathogenesis is unclear. Inflammation at the maternal‐fetal interface in preterm labor leads to the infiltration of neutrophils, which promotes inflammatory responses and induces the degradation of extracellular matrix and cell apoptosis, thus contributing to preterm labor. It is unclear whether neutrophil extracellular traps (NETs), a functional form of neutrophils, are involved in preterm labor.

Keywords: preterm labor; cell; cell apoptosis; promote ros; nets promote

Journal Title: American Journal of Reproductive Immunology
Year Published: 2022

Link to full text (if available)


Share on Social Media:                               Sign Up to like & get
recommendations!

Related content

More Information              News              Social Media              Video              Recommended



                Click one of the above tabs to view related content.