In the current issue of Acta Physiologica, Guessoum et al. address a question that has intrigued investigators for decades within kidney and blood pressure research fields1 . In conditions where… Click to show full abstract
In the current issue of Acta Physiologica, Guessoum et al. address a question that has intrigued investigators for decades within kidney and blood pressure research fields1 . In conditions where the renin-angiotensin aldosterone system (RAAS) is chronically challenged, the renin-producing cells increase significantly in number and their localization expands from a juxtaglomerular position to a widespread distribution upstream the afferent glomerular arterioles and to extraglomerular mesangial cells.
               
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