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BK polyoma virus (BKPyV) associated nephropathy (BKPyVAN) is a major cause of kidney graft loss in renal transplant patients. Interferons (IFNs) are an important innate immune response against viral infections… Click to show full abstract
BK polyoma virus (BKPyV) associated nephropathy (BKPyVAN) is a major cause of kidney graft loss in renal transplant patients. Interferons (IFNs) are an important innate immune response against viral infections and genetic polymorphisms of the IFN‐pathways can affect susceptibility and mortality during viral infection. Here, we investigated whether the dinucleotide polymorphism rs368234815 (ΔG/TT) in the IFNL4 gene contributed to BKPyV reactivation or BKPyVAN after living‐donor kidney transplantation.
Keywords:
polymorphism;
kidney;
donor kidney;
associated nephropathy;
virus;
living donor
Journal Title: Clinical Transplantation
Year Published: 2022
Link to full text (if available)
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Journal Title: Clinical Transplantation
Year Published: 2022
Link to full text (if available)
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recommendations!