Pathophysiological evidences on ischemic stroke (IS) indicate a critical involvement of post-ischemic inflammation in determining neuronal injury. In last decades, several inflammatory molecules have been investigated as potential diagnostic and/or… Click to show full abstract
Pathophysiological evidences on ischemic stroke (IS) indicate a critical involvement of post-ischemic inflammation in determining neuronal injury. In last decades, several inflammatory molecules have been investigated as potential diagnostic and/or prognostic tools. At present, however, no specific pathophysiological factor has been identified for being recommended in the clinical management of the disease. This article is protected by copyright. All rights reserved.
               
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