LAUSR

We investigated the effect of in situ stresses on fluid flow in a natural fracture network. The fracture network model is based on an actual critically connected (i.e., close to the percolation threshold) fracture pattern mapped from a field outcrop. We derive stress-dependent fracture aperture fields using a hybrid finite-discrete element method. We analyze the changes of aperture distribution and fluid flow field with variations of in situ stress orientation and magnitude. Our simulations show that an isotropic stress loading tends to reduce fracture apertures and suppress fluid flow, resulting in a decrease of equivalent permeability of the fractured rock. Anisotropic stresses may cause a significant amount of sliding of fracture walls accompanied with shear-induced dilation along some preferentially oriented fractures, resulting in enhanced flow heterogeneity and channelization. When the differential stress is further elevated, fracture propagation becomes prevailing and creates some new flow paths via linking preexisting natural fractures, which attempts to increase the bulk permeability but attenuates the flow channelization. Comparing to the shear-induced dilation effect, it appears that the propagation of new cracks leads to a more prominent permeability enhancement for the natural fracture system. The results have particularly important implications for predicting the hydraulic responses of fractured rocks to in situ stress fields and may provide useful guidance for the strategy design of geofluid production from naturally fractured reservoirs.