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A paralog synthetic–lethal interaction between VRK1 and VRK2 sensitizes VRK2-methylated glioblastoma to perturbation of VRK1 kinase activity, supporting VRK1 as a drug discovery target in this disease. Click to show full abstract
A paralog synthetic–lethal interaction between VRK1 and VRK2 sensitizes VRK2-methylated glioblastoma to perturbation of VRK1 kinase activity, supporting VRK1 as a drug discovery target in this disease.
Keywords:
synthetic lethal;
glioblastoma;
lethal target;
vrk1 synthetic;
target vrk2
Journal Title: Cancer Research
Year Published: 2022
Link to full text (if available)
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Journal Title: Cancer Research
Year Published: 2022
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!