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Transgenic knockin mice expressing a common loss-of-function mutation in human TET2 exhibit aging-related accelerated myeloid leukemia development and skewing of myelopoiesis toward the production of proinflammatory MHC-IIhi monocytes that may… Click to show full abstract
Transgenic knockin mice expressing a common loss-of-function mutation in human TET2 exhibit aging-related accelerated myeloid leukemia development and skewing of myelopoiesis toward the production of proinflammatory MHC-IIhi monocytes that may contribute to disease. See related article by Yeaton et al., p. 2392 (2).
Keywords:
dangerous liaisons;
inflammatory monocytes;
liaisons tet2;
tet2 mutation;
mutation;
mutation inflammatory
Journal Title: Cancer discovery
Year Published: 2022
Link to full text (if available)
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Journal Title: Cancer discovery
Year Published: 2022
Link to full text (if available)
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recommendations!