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We read with interest the study by Joffre and colleagues, “COVID-19associated LungMicrovascular Endotheliopathy: A “From the Bench” Perspective” (1). The authors propose a model of endotheliopathy due to both endothelial… Click to show full abstract
We read with interest the study by Joffre and colleagues, “COVID-19associated LungMicrovascular Endotheliopathy: A “From the Bench” Perspective” (1). The authors propose a model of endotheliopathy due to both endothelial cell severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection as well as inflammatory cytokines that likely contribute to endothelial dysfunction. In support of this model, we have previously documented that both SARS-CoV2 endothelial cell infection and the infection-associated immune activation contribute to
Journal Title: American Journal of Respiratory and Critical Care Medicine
Year Published: 2022
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