In this issue of Blood , [Beck et al][1][1][2] describe adenosine diphosphate (ADP)–induced responses in the platelet phosphoproteome, which are overridden by the prostacyclin analog iloprost, revealing a hidden complexity… Click to show full abstract
In this issue of Blood , [Beck et al][1][1][2] describe adenosine diphosphate (ADP)–induced responses in the platelet phosphoproteome, which are overridden by the prostacyclin analog iloprost, revealing a hidden complexity of signal transduction that determines functional outcome. ![Figure][3]
               
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