In this issue of Blood, Baaten et al report a new method of comparing published murine studies assessing the impact of single gene defects on arterial thrombus formation, thromboembolism, and… Click to show full abstract
In this issue of Blood, Baaten et al report a new method of comparing published murine studies assessing the impact of single gene defects on arterial thrombus formation, thromboembolism, and tail bleeding time. This approach is meant to obtain reliable information on the role of specific genes in thrombosis and hemorrhage in comparison with human orthologs.1
               
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