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Key Points Glofitamab induced dose-dependent induction of cytokines and T-cell margination, proliferation, and activation in peripheral blood. Tumor cell intrinsic factors (eg, TP53 and MYC signaling) are associated with resistance… Click to show full abstract
Key Points Glofitamab induced dose-dependent induction of cytokines and T-cell margination, proliferation, and activation in peripheral blood. Tumor cell intrinsic factors (eg, TP53 and MYC signaling) are associated with resistance to glofitamab.
Keywords:
pharmacodynamics molecular;
molecular correlates;
correlates response;
glofitamab;
response glofitamab;
glofitamab relapsed
Journal Title: Blood Advances
Year Published: 2021
Link to full text (if available)
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Journal Title: Blood Advances
Year Published: 2021
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!