LAUSR

EJD, vol. 27, n◦ 6, November-December 2017 PG associated with arteriovenous dialysis shunts have been reported to date [6, 7]. We speculate that the debridement for the stasis ulcers or the transcatheter arterial microembolization provoked PG in our case. It is known that PG rarely occurs at the sites of surgical incision, which is described as postoperative PG. Operative trauma and pathergy seem to induce postoperative PG through up-regulation of neutrophils [1]. Pathergy is considered as a condition of altered tissue reactivity occurring in response to the trauma [8]. Although surgical debridement probably induced PG through pathergy, we cannot exclude the possibility that transcatheter arterial microembolization provoked this PG, although no reports have been published regarding an association between PG and transcatheter arterial microembolization. Since transcatheter arterial microembolization using imipenem/cilastatin sodium is a recently proposed approach for management [9], the number of cases treated in this way will increase as will those of PG using this method.