LAUSR

127 the smaller one (17 × 10, near the lateral site of the tricuspid anulus) were connected with RV aneurysms. Apixaban (5 mg twice daily) was in‐ troduced. Daily echocardiography showed grad‐ ual resolution of thrombi and their disappear‐ ance after 18 days, confirmed by CT (FIGURE 1F–1H). ARVC is an inherited disease characterized by progressive fibrofatty replacement of the RV myocardium resulting in RV dilation, hypokine‐ sis, and development of local aneurysms, with associated arrhythmias originating in the RV. Such arrhythmias may lead to sudden cardiac death. The estimated prevalence of all thrombo‐ embolic complications in ARVC is 2% to 4% of pa‐ tients.1,2 In some patients, pulmonary emboliza‐ tion from right ‐sided intracardiac thrombi may be responsible for an unfavorable course of the dis‐ ease. In multimodality imaging of RV thrombi by echocardiography, MRI, or CT, the availability of A 30 ‐year ‐old man with previously diagnosed ar‐ rhythmogenic right ventricular cardiomyopathy (ARVC) and with a family history of the disease was admitted due to hemodynamically unstable ventricular tachycardia (VT). Sinus rhythm was restored by electrical cardioversion. Echocardio‐ graphic evaluation was consistent with magnet‐ ic resonance imaging (MRI) performed several months earlier, which showed right ventricular (RV) enlargement with multiple local aneurysms. No intracardiac thrombi were seen at that time (FIGURE 1A and 1B). Two days later, the patient un‐ derwent implantable cardioverter ‐defibrillator implantation. On the second postprocedural day, 2 RV thrombi were found on control echocar‐ diography and visualized on computed tomog‐ raphy (CT) performed to exclude pulmonary embolism (FIGURE 1C–1E). Both the larger throm‐ bus (29 × 13 mm, in the RV outflow tract) and CLINICAL IMAGE