The Editor-in-Chief and Editorial Board of Clinical Hemorheology and Microcirculation (CHM), as well as the Publisher (IOS Press) have decided to set an annual prize, named the A.L. Copley Best… Click to show full abstract
The Editor-in-Chief and Editorial Board of Clinical Hemorheology and Microcirculation (CHM), as well as the Publisher (IOS Press) have decided to set an annual prize, named the A.L. Copley Best Paper Prize, to recognize the best article published every year in CHM beginning in 2016. This prize has been named in honor of the Journal’s founding editor, Alfred Lewin Copley. Dr. A.L. Copley was a German American medical scientist who introduced the term “Hemorheology” and defined this area of science.Firstofall, the editorial team wish to thank all authors for their valuable contributions in 2020. A group of three editors was elected by the editorial board to select the best paper in a multistep process. The criteria for prize selection included: originality and innovation, theoretical contribution, clarity of writing and presentation, and expected impact. In the first step, each of the three editors listed the 10 best papers separately. From these 30 papers the prize committee looked for and structural compensatory mechanisms of a chronic kidney disease rat model.” The authors studied two groups of female Sprague-Dawley rats: one subjected to nephrectomy and one sham-operated group where no intervention was made. The study revealed that serum creatinine increased in the nephrectomy group ( p = 0.008), accompanied with a decrease of red blood cell count ( p = 0.028) and hemoglobin concentration ( p = 0.015). Erythrocyte aggregation param-eters slightly increased in nephrectomy group, while red blood cell deformability did not show any significant changes. The microcirculation remained intact in the remnant kidney of the nephrectomy group. However, in comparison with the sham-operated group, the diameter of glomeruli increased significantly ( p < 0.01). The authors concluded that erythrocyte mass was influenced more than micro-rheological properties in this model. The main compensation mechanism was rather structural than at microcirculatory level.
               
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