Myocardial fibrosis (MF) is a hallmark pathological outcome of many cardiovascular diseases and a key component of cardiac remodeling. The p38 MAPK, JNK, and ERK signaling pathways are central to… Click to show full abstract
Myocardial fibrosis (MF) is a hallmark pathological outcome of many cardiovascular diseases and a key component of cardiac remodeling. The p38 MAPK, JNK, and ERK signaling pathways are central to this process. This review summarizes the roles and interactions of these factors in MF and identifies relevant drug and non-pharmacological therapies targeting these pathways.
Keywords:
research progress;
fibrosis;
myocardial fibrosis;
mapk signaling;
progress mapk;
mechanism research
Journal Title: Frontiers in Cardiovascular Medicine
Year Published: 2025
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Journal Title: Frontiers in Cardiovascular Medicine
Year Published: 2025
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!