LAUSR.org creates dashboard-style pages of related content for over 1.5 million academic articles. Sign Up to like articles & get recommendations!

MicroRNA-24 attenuates diabetic vascular remodeling by suppressing the NLRP3/caspase-1/IL-1β signaling pathway

Photo by jessbaileydesigns from unsplash

Subsequently to the publication of the above article, the authors have realized that the immunohistochemical staining of CD31 in the 'Ad‑NC group' portrayed in Fig. 7 on p. 1539 was published with… Click to show full abstract

Subsequently to the publication of the above article, the authors have realized that the immunohistochemical staining of CD31 in the 'Ad‑NC group' portrayed in Fig. 7 on p. 1539 was published with an image that had been incorrectly captured from the 'Saline' group. This inadvertent error arose as a consequence of miscommunication between a pair of the authors on the paper when capturing the images. The authors have re‑examined their original data and identified the correct data for the Ad‑NC group in Fig. 7. The corrected version of Fig. 7, showing the correct CD31 staining data for the Ad‑NC group, is shown below. Note that this error did not affect the major conclusions reported in the paper. All the authors have agreed to this corrigendum, and apologize to the Editor of International Journal of Molecular Medicine and to the readership for any inconvenience caused. [the original article was published in International Journal of Molecular Medicine 45: 1534‑1542, 2020; DOI: 10.3892/ijmm.2020.4533].

Keywords: journal molecular; group; molecular medicine; medicine; microrna attenuates; international journal

Journal Title: International Journal of Molecular Medicine
Year Published: 2022

Link to full text (if available)


Share on Social Media:                               Sign Up to like & get
recommendations!

Related content

More Information              News              Social Media              Video              Recommended



                Click one of the above tabs to view related content.