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Author's summary AC005332.7 overexpression alleviated myocardial injury via suppressing ferroptosis in vitro and in vivo. AC005332.7 could directly bind to miR-331-3p to promote cyclin D2 (CCND2) expression. MiR-331-3p sufficiency or… Click to show full abstract
Author's summary AC005332.7 overexpression alleviated myocardial injury via suppressing ferroptosis in vitro and in vivo. AC005332.7 could directly bind to miR-331-3p to promote cyclin D2 (CCND2) expression. MiR-331-3p sufficiency or CCND2 knockdown reversed AC005332.7-mediated inhibition of ferroptosis in oxygen and glucose-deprivation-disposed AC16 cells. CCND2 was highly expressed when overexpressed AC005332.7 in acute myocardial infarction.
Keywords:
mir 331;
ac005332;
ccnd2;
myocardial infarction;
acute myocardial;
ferroptosis
Journal Title: Korean Circulation Journal
Year Published: 2023
Link to full text (if available)
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Journal Title: Korean Circulation Journal
Year Published: 2023
Link to full text (if available)
Share on Social Media: Sign Up to like & get
recommendations!