LAUSR

The recovery course of patients with Guillain-Barre syndrome (GBS) can be complicated by non-neurological systemic perturbations. Autonomic dysfunction associated with GBS affects up to two-thirds of patients and can lead to a reduction in the counterregulatory catecholamine response. This increases the risk of severe hypoglycaemia, especially in diabetics.[1] Sudden, severe hypoglycaemia can damage both white and gray matter of the cerebrum resulting in encephalopathy.[2] Attention to glycaemic control is an extremely crucial component in the management of these patients. Due to associated autonomic instability, the warning signs of fatal hypoglycaemia may go unnoticed,[3] like the index case discussed herein. The patient was not a diabetic,