LAUSR

The authors presented a case of an extensive dermatophytosis that simulated clinically, histologically, and immunologically a bullous pemphigoid. The authors concluded that the possible mechanism would be a molecular mimicry that could trigger a hypersensitive response from the stratum corneum to the dermoepidermal junction and try to make a distinction with previous reports of bullous tinea corporis. We agree with the authors that it is not a clinical variety of this last entity since the cases of tinea bullosa referred by the authors are completely different in several aspects. The cases of bullous tinea corporis commonly present fungi in the blister cavity, also have a more rapid evolution (around a week or less) and are frequently localized, while the cases of bullous pemphigoid are chronic and generally disseminated; bullous tinea has intraepidermal blisters, while pemphigoid develops subepidermal blisters. Moreover, there are no deposits of antibodies to direct immunofluorescence in the former, an aspect frequently seen in the latter.