LAUSR

Calcinosis cutis is a term used to describe a group of disorders characterized by deposition of calcium salts in the skin and subcutaneous tissue. Virchow initially described calcinosis cutis in 1855.[1] Calcinosis cutis is classified into four major types according to etiology: dystrophic, metastatic, iatrogenic, and idiopathic.[1] Dystrophic calcinosis is calcification associated with infection, inflammatory processes, cutaneous neoplasm, or connective tissue diseases.[2,3] Metastatic calcification results from elevated serum levels of calcium or phosphorus.[4] Iatrogenic and traumatic calcinosis are those types which are associated with medical procedures.[4] Idiopathic calcinosis cutis is cutaneous calcification of unknown cause with normal serum calcium, no history of local tissue injury and systemic metabolic disorder.[5] Subepidermal calcified nodule and tumoral calcinosis are idiopathic forms of calcification. A few rare types have been variably classified as dystrophic or idiopathic.[6] These include calcinosis cutis circumscripta, calcinosis cutis universalis, tumoral calcinosis, and transplant‐associated calcinosis cutis.[6] Calcinosis cutis with Raynaud’s phenomenon, esophageal dysmotility, sclerodactyly, and telangiectasia is referred to as CREST syndrome.[7‐9] Here, we present a case of idiopathic calcinosis cutis over back in a 17‐year‐old young male along with the histopathological correlation.