An Akt3 Splice Variant Lacking the Serine 472 Phosphorylation Site Promotes Apoptosis and Suppresses Mammary Tumorigenesis.
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DOI: 10.1158/0008-5472.can-15-1462
Abstract: The Akt pathway is a well-known promoter of tumor malignancy. Akt3 is expressed as two alternatively spliced variants, one of which lacks the key regulatory serine 472 phosphorylation site. Whereas the function of full-length Akt3… read more here.
Keywords: akt3 s472; akt3; 472 phosphorylation; phosphorylation site ... See more keywords