Excessive/Aberrant and Maladaptive Synaptic Plasticity: A Hypothesis for the Pathogenesis of Alzheimer’s Disease
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DOI: 10.3389/fnagi.2022.913693
Abstract: The amyloid hypothesis for the pathogenesis of Alzheimer’s disease (AD) is widely accepted. Last year, the US Food and Drug Administration considered amyloid-β peptide (Aβ) as a surrogate biomarker and approved an anti-Aβ antibody, aducanumab,… read more here.
Keywords: pathogenesis alzheimer; hypothesis; alzheimer disease; hypothesis pathogenesis ... See more keywords