Oral ethinylestradiol-levonorgestrel normalizes fructose-induced hepatic lipid accumulation and glycogen depletion in female rats.
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DOI: 10.1139/cjpp-2019-0037
Abstract: The present study investigated effects of oral ethinylestradiol-levonorgestrel (EEL) on hepatic lipid and glycogen contents during high fructose (HF) intake and determined whether pyruvate dehydrogenase kinase-4 (PDK-4) and Glucose-6-phosphate dehydrogenase (G6PD) activity were involved in… read more here.
Keywords: hepatic lipid; lipid accumulation; accumulation glycogen; glycogen ... See more keywords