Interleukin-35 stimulates tumor necrosis factor-α activated osteoblasts differentiation through Wnt/β-catenin signaling pathway in rheumatoid arthritis.
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DOI: 10.1016/j.intimp.2019.105810
Abstract: Interleukin (IL)-35 plays an important role in the pathogenesis of rheumatoid arthritis (RA), which is characterized by tumor necrosis factor (TNF)-α activated bone loss beginning early and persisting over time. The aim of this study… read more here.
Keywords: catenin signaling; tnf activated; signaling pathway; activated osteoblasts ... See more keywords