Amyloid-β Oligomers Regulate ADAM10 Synaptic Localization Through Aberrant Plasticity Phenomena
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DOI: 10.1007/s12035-019-1583-5
Abstract: A disintegrin and metalloproteinase 10 (ADAM10) is a synaptic enzyme that has been previously shown to limit amyloid-β1–42 (Aβ1–42) peptide formation in Alzheimer’s disease (AD). Furthermore, ADAM10 participates to spine shaping through the cleavage of… read more here.
Keywords: synaptic localization; plasticity phenomena; adam10 synaptic; aberrant plasticity ... See more keywords