miR‐192‐5p mediates hypoxia/reoxygenation‐induced apoptosis in H9c2 cardiomyocytes via targeting of FABP3
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DOI: 10.1002/jbt.21873
Abstract: Myocardial ischemia/reperfusion (I/R) injury is a leading cause of morbidity and mortality. In this study, we investigated the role of miR‐192‐5p in hypoxia/reoxygenation (H/R)‐induced cardiomyocyte apoptosis. H9c2 cardiomyocytes were subjected to H/R and tested for… read more here.
Keywords: mir 192; apoptosis h9c2; hypoxia reoxygenation; apoptosis ... See more keywords
The protective effect of lipid emulsion in preventing bupivacaine-induced mitochondrial injury and apoptosis of H9C2 cardiomyocytes
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DOI: 10.1080/10717544.2016.1261379
Abstract: Abstract Lipid emulsion (LE) has been shown to be effective in the resuscitation of bupivacaine-induced cardiac arrest, but the precise mechanism of this action has not been fully elucidated. Pursuant to this lack of information… read more here.
Keywords: bupivacaine induced; h9c2 cardiomyocytes; bupivacaine; apoptosis h9c2 ... See more keywords
Apigenin suppresses the apoptosis of H9C2 rat cardiomyocytes subjected to myocardial ischemia-reperfusion injury via upregulation of the PI3K/Akt pathway
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DOI: 10.3892/mmr.2018.9115
Abstract: Apigenin, a flavonoid with multiple physiological and pharmacological activities, is associated with the prevention of cardiovascular diseases. The present study aimed to examine the roles and mechanisms of apigenin in the apoptosis of H9C2 rat… read more here.
Keywords: h9c2 rat; apigenin; apoptosis h9c2; injury ... See more keywords
MicroRNA-194-5p attenuates hypoxia/reoxygenation-induced apoptosis in H9C2 cardiomyocytes by inhibiting the over-activation of RAC1 protein
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DOI: 10.3892/mmr.2022.12920
Abstract: Ras-related C3 botulinum toxin substrate 1 (RAC1), a member of the Rac family of guanosine triphosphate phosphohydrolases, has been suggested to be a regulator of myocardial injury during ischemia and reperfusion (I/R). Whether microRNAs (miRs)… read more here.
Keywords: apoptosis h9c2; hypoxia reoxygenation; apoptosis; rac1 ... See more keywords