The drinking water contaminant dibromoacetonitrile delays G1-S transition and suppresses Chk1 activation at broken replication forks
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DOI: 10.1038/s41598-017-13033-8
Abstract: Chlorination of drinking water protects humans from water-born pathogens, but it also produces low concentrations of dibromoacetonitrile (DBAN), a common disinfectant by-product found in many water supply systems. DBAN is not mutagenic but causes DNA… read more here.
Keywords: water; broken replication; delays transition; replication forks ... See more keywords