BNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway
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DOI: 10.1038/s41598-017-01936-5
Abstract: Fibrosis is an important, structurally damaging event that occurs in pathological cardiac remodeling, leading to cardiac dysfunction. BNIP3L is up-regulated in pressure overload-induced heart failure and has been reported to play an important role in… read more here.
Keywords: fibrosis; ca2 tgf; cardiac fibrosis; bnip3l ... See more keywords