A Novel Somatic Mutation of CACNA1H p.V1937M in Unilateral Primary Hyperaldosteronism
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DOI: 10.3389/fendo.2022.816476
Abstract: Background Somatic mutations for excess aldosterone production have been frequently identified as important roles in the pathogenesis of unilateral primary hyperaldosteronism (uPA). Although CACNA1H mutation represents a minor etiology in primary aldosteronism, it plays a… read more here.
Keywords: cacna1h v1937m; unilateral primary; primary hyperaldosteronism; novel somatic ... See more keywords