CCN2 deficiency in smooth muscle cells triggers cell reprogramming and aggravates aneurysm development
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DOI: 10.1172/jci.insight.162987
Abstract: Vascular smooth muscle cell (SMC) phenotypic switching is widely recognized as a key mechanism responsible for the pathogenesis of several aortic diseases, such as aortic aneurysm. Cellular communication network factor 2 (CCN2), often upregulated in… read more here.
Keywords: smooth muscle; ccn2; smc; ccn2 deficiency ... See more keywords