Mycobacterium tuberculosis Heat-Shock Protein 16.3 Induces Macrophage M2 Polarization Through CCRL2/CX3CR1
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DOI: 10.1007/s10753-019-01132-9
Abstract: Mycobacterium tuberculosis, the pathogen of tuberculosis (TB), can survive in host macrophages and induce macrophages to M2 phenotype might result in latent MTB infection. During the latent phase, the expression of MTB heat-shock protein 16.3… read more here.
Keywords: ccrl2 cx3cr1; polarization; hsp16; tuberculosis ... See more keywords