Id2 Represses Aldosterone-Stimulated Cardiac T-Type Calcium Channels Expression
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DOI: 10.3390/ijms22073561
Abstract: Aldosterone excess is a cardiovascular risk factor. Aldosterone can directly stimulate an electrical remodeling of cardiomyocytes leading to cardiac arrhythmia and hypertrophy. L-type and T-type voltage-gated calcium (Ca2+) channels expression are increased by aldosterone in… read more here.
Keywords: type; channels expression; aldosterone stimulated; id2 ... See more keywords