Chemical denervation using botulinum toxin increases Akt expression and reduces submaximal insulin-stimulated glucose transport in mouse muscle.
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DOI: 10.1016/j.cellsig.2018.10.014
Abstract: Botulinum toxin A (botox) is a toxin used for spasticity treatment and cosmetic purposes. Botox blocks the excitation of skeletal muscle fibers by preventing the release of acetylcholine from motor nerves, a process termed chemical… read more here.
Keywords: chemical denervation; insulin; expression; glucose transport ... See more keywords