YB-1 increases glomerular, but decreases interstitial fibrosis in CNI-induced nephropathy.
Sign Up to like & getrecommendations! Published in 2018 at "Clinical immunology"
DOI: 10.1016/j.clim.2018.07.002
Abstract: Calcineurin inhibitors (CNIs) are a cornerstone of the current treatment in solid organ transplantation and autoimmune disease. However, CNIs also bear deleterious effects as they cause glomerular and tubulointerstitial fibrosis in the kidney. We recently… read more here.
Keywords: cni; fibrosis; increases glomerular; glomerular decreases ... See more keywords
CD44 as a pathological marker for the early detection of calcineurin inhibitor-induced nephrotoxicity post kidney transplantation.
Sign Up to like & getrecommendations! Published in 2022 at "Biomedical research"
DOI: 10.2220/biomedres.43.181
Abstract: Long-term calcineurin inhibitor (CNI) administration causes irreversible nephrotoxicity. Therefore, early CNI-induced nephrotoxicity detection is necessary for patients who will need long-term CNI administration. There is no pathological indicator for early CNI-induced nephrotoxicity. Here, serial protocol… read more here.
Keywords: cni induced; kidney; cni; detection ... See more keywords