Reciprocal interactions among Cobll1, PACSIN2, and SH3BP1 regulate drug resistance in chronic myeloid leukemia
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DOI: 10.1002/cam4.4727
Abstract: Cobll1 affects blast crisis (BC) progression and tyrosine kinase inhibitor (TKI) resistance in chronic myeloid leukemia (CML). PACSIN2, a novel Cobll1 binding protein, activates TKI‐induced apoptosis in K562 cells, and this activation is suppressed by… read more here.
Keywords: pacsin2 sh3bp1; resistance; pacsin2; cobll1 pacsin2 ... See more keywords