TLR2/4 promotes PGE2 production to increase tissue damage in Escherichia coli-infected bovine endometrial explants via MyD88/p38 MAPK pathway.
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DOI: 10.1016/j.theriogenology.2020.04.004
Abstract: Prostaglandin E2 (PGE2), a lipid mediator, is released by several cell types including endometrial cells and plays a central role in bacterial infection of the endometrium during inflammation. PGE2 production accumulated in Escherichia coli (E. coli)… read more here.
Keywords: coli infected; endometrial tissue; tissue damage; tissue ... See more keywords