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CTRP3 protected against doxorubicin-induced cardiac dysfunction, inflammation and cell death via activation of Sirt1.

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0 Published in 2018 at "Journal of molecular and cellular cardiology"

DOI: 10.1016/j.yjmcc.2017.10.008

Abstract: BACKGROUND Inflammation and myocytes apoptosis play critical roles in the development of doxorubicin (DOX)-induced cardiotoxicity. Our previous study found that C1q/tumour necrosis factor-related protein-3 (CTRP3) could inhibit cardiac inflammation and apoptosis of myocytes but its… read more here.

Keywords: sirt1; dox induced; ctrp3 protected; inflammation ... See more keywords