Truncation of CXCL8 to CXCL8(9‐77) enhances actin polymerization and in vivo migration of neutrophils
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DOI: 10.1002/jlb.3ab0220-470r
Abstract: CXCL8 is the principal human neutrophil‐attracting chemokine and a major mediator of inflammation. The chemokine exerts its neutrophil‐chemotactic and neutrophil‐activating activities via interaction with glycosaminoglycans (GAGs) and activation of the G protein‐coupled receptors (GPCRs) CXCR1… read more here.
Keywords: truncation; cxcl8 cxcl8; actin polymerization; nh2 terminal ... See more keywords